Syndrome X (metabolic syndrome) is a serious metabolic disorder linked to a group of separate symptoms including abdominal “apple shape” body image, glucose intolerance, insulin resistance, elevated blood pressure, atherogenic dyslipidemia, disrupted cholesterol panel, leading to an increased risk of heart attack, stroke, diabetes, gout, kidneys failure, Alzheimer’s disease, and many others. (Source, Wiki)
Unfortunately metabolic syndrome is on the rise, already recognized as the newest global pandemic of the 21st century. (Grundy, 2008) Except the above mentioned features, there is another major marker linked to the metabolic syndrome cluster, known under the term “fatty liver.” As obesity rates continue to climb throughout the Western world, fatty liver disease is becoming the second major factor linked to the metabolic syndrome cluster.
Liver steatosis has long been associated with long-term, excessive alcohol consumption, but there is another “non-alcoholic” version of this disease that is believed to be caused by poor diet, loaded with simple sugars and complex carbohydrates. Called a “non-alcoholic fatty liver disease” (NAFLD), or liver steatosis, the disorder affects about a third of the American adult population and its numbers are progressively rising.
Fatty liver disease is the buildup of fat in the liver cells. As the name suggests the liver is becoming “fatty” when its liver cells (hepatocytes) accumulate too much fat, finally impacting its ability to function. The above disorder is also called liver steatosis or non-alcoholic fatty liver (NAFLD) disease. Fat accumulations may also lead to chronic inflammation (hepatitis) and cirrhosis (scarring) of the liver, finally progressing to terminal liver failure.
Non-alcoholic fatty liver disease (NAFLD) is actually the most common form of chronic liver disorder quite common to for the Western industrialized world. NAFLD comes gradually albeit silently. Patients are simply unaware that they have the above problem, as it rarely causes symptoms itself, but when accelerates- it can lead to serious liver damage and to final death. Often there are no symptoms, despite some sense of fatigue or light pain in the upper right abdominal area may appear. In case you notice that your belt feels tighter, consider a visit at your doctor office, gain some knowledge on the syndrome, and get some proof that the disease is not in your genes. (Source, Wiki)
Although it takes a long time of chronic overeating and consuming “nutrition empty” — “caloric dense” diet with chronic immobilization, it seems pretty much that your fork works as your main tool that may build your health and figure, or to bring the disease back to your life.
Accordingly, in older men and women, Syndrome X is associated with excess accumulation of visceral abdominal or intramuscular fat, even in completely “non-obese” normal-weight individuals (Bays & Dujovne, 2006). The authors state that the flux of nonesterified fatty acids to the liver is tied into visceral fat accumulation as a contributing factor. They believe that, “Fat dysfunction (adiposopathy) is a more rational treatment target for metabolic disease than obesity alone. This may occur through appropriate diet, physical exercise, and other lifestyle changes, and/or from drug therapies.”
A research done by a team (Phillips, Jing & Heymsfield. 2008) proved that it is the visceral fat the major factor that underlies the metabolic syndrome appearance in women, instead of the insulin resistance phenomena, once belived to be the main culprit behind the syndrome. Actually, it is already proven that visceral obesity causes insulin resistance, but it is not related to high blood pressure.
Another study published in the European Journal of Gastroenterology & Hepatology (2006) found that for a period of three months correct nutrition, combined with one-hour exercise daily helped obese teens with liver steatosis to improve their status of fatty liver disease. As a result of the above study, 29 percent of all the participants reduced their liver symptoms and 50 percent lost their weight.
The above cited studies proved once again that the treatment and prevention of NAFLD should be based on correct nutrition, consuming nutritionally healthy and balanced diet, rapid weight loss through correct diet and exercise, consuming diet with lower glycemic index, daily exercise, and additional supplementation of dietary vitamins and supplements, while avoiding alcohol, sugars and environmental toxicities.
Obviously, the concept of metabolic syndrome treatment and prevention extends beyond the existing knowledge on how to treat its sepаrаte symptoms. Effective prevention аnd treаtment of metаbolic syndrome should involve а multifаceted/ multidimensional аpproаch directed to аll of its cаrdinаl components and markers. (Holt, 2002)
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